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Breed information
Length range : 69-81 cm
Height range : 36-43cm
Weight range : 8-14kg
Coat : Long double coat with fine outer coat and woolly undercoat
Life expectancy years : 12-16
Colors: wide range of colors.
Activitity Level : Moderate
Origin: Tibet
Similar Breeds : Bearded Collie|Lhasa Apso|Polish Lowland Sheepdog
History
The breed traces back thousands of years to Tibetan monasteries where it served as a companion and watchdog and was associated with Buddhist monks. Modern breed history began in 1922 when Dr. Agnes Grieg received a Tibetan Terrier in northern India and later established the breed line recognized by kennel clubs. The AKC recognized the Tibetan Terrier in 1973.
Temperament
affectionate|sensitive|clever|loyal|alert
Personality
affectionate|loyal|friendly|confident|alert|intelligent|playful|sensitive
Needs
a lot of grooming|regular exercise|early socialization|positive training|companionship|routine eye monitoring|coat maintenance to prevent mats
Social Needs
strongly devoted to family|reserved with strangers until familiar|does best with regular human company|can be good with children and other dogs when properly introduced|may not do well alone for long
Grooming
High grooming needs. If kept long the coat needs frequent to daily brushing and combing to prevent matting. Weekly ear cleaning , routine nail care and baths as needed are recommended and many pet owners keep the coat trimmed shorter for easier care.
Nutrition
Feed a high-quality AAFCO-compliant diet for small- or medium-sized breeds and appropriate life stage. Adults are commonly fed twice daily and puppies three to four times daily. Treats should stay limited and supplements are generally only used when a veterinarian recommends them for skin or joint concerns.
Breed Standard
The Tibetan Terrier is a sturdy, medium-sized, square-built dog with a profuse double coat and unique large, flat “snowshoe” feet. Standing 14 to 16 inches tall, they possess an alert, loyal, and good-natured temperament, functioning historically as companions and watchdogs rather than true terriers.
Did you know?
Despite the name the Tibetan Terrier is not a true terrier. It is known as the Holy Dog of Tibet and its broad snowshoe feet helped it move across snowy mountain terrain.
Most Common / iconic illnesses
Progressive Retinal Atrophy, Primary Lens Luxation, Hip Dysplasia, Cataracts, Neuronal Ceroid Lipofuscinosis
Symptoms
Progressive Retinal Atrophy
1. Night blindness 2. Reluctance to enter dark areas 3. Hesitance on stairs 4. Dilated pupils with reflective eyes 5. Bumping into objects or eventual blindness may occur. || Primary Lens Luxation
Primary Lens Luxation
1. Red painful eye 2. Squinting 3. Increased tearing 4. Hazy or cloudy eye 5. Sudden vision loss or enlarged eye may occur. || Hip Dysplasia
Hip Dysplasia
1. Bunny-hopping gait 2. Difficulty rising 3. Limping or stiffness 4. Reluctance to run , jump or use stairs 5. Decreased hindlimb muscle mass or hip pain may occur. || Cataracts
Cataracts
1. Cloudy or white lens appearance 2. Blurry vision 3. Bumping into objects 4. Hesitation in dim light 5. Progressive vision loss may occur. || Neuronal Ceroid Lipofuscinosis
Neuronal Ceroid Lipofuscinosis
1. Aggression or anxiety 2. Uncoordinated gait 3. Stumbling or crossing over 4. Vision changes especially in dim light 5. Seizures , loss of house training or difficulty with stairs may occur.
Onset of symptoms
Progressive Retinal Atrophy
It usually begins during adulthood and night vision is often affected first before vision loss gradually progresses.
Probability Of Onset:
Moderate to high breed-specific risk is present because Tibetan Terriers are listed among breeds with known PRA predisposition.
Primary Lens Luxation
It most commonly develops between 3 and 8 years of age and structural eye changes may be evident by about 20 months.
Probability Of Onset:
Moderate to high breed-specific risk is present because the condition is inherited and prompt management is emphasized in predisposed breeds.
Neuronal Ceroid Lipofuscinosis
It typically becomes clinically evident in early to mid-adulthood and breed-matched external text places onset around 4 to 6 years of age.
Probability Of Onset:
Moderate to high breed-specific risk is present because the OFA provides a breed-specific DNA test for Tibetan Terriers and the disease is inherited.
Cataracts
They can develop at any age. Congenital cases are present at birth while inherited or diabetic cataracts may develop earlier and age-related cataracts may appear later in life.
Probability Of Onset:
Moderate risk is present because cataracts are a recognized ocular issue in Tibetan Terriers and may also occur secondary to diabetes or other eye disease.
Hip Dysplasia
Signs may appear as early as a few months old or later in life depending on severity and arthritis development.
Probability Of Onset:
Moderate risk is present because it is a recognized inherited orthopedic concern in the breed.
Reasons
Progressive Retinal Atrophy
Normal retinal photoreceptor integrity is lacking because inherited genetic variants cause degeneration of rods and cones. No specific nutrient deficiency is identified as the direct cause in the provided texts. Rod loss affects night vision first and progressive cell loss leads toward blindness. The disorder is non-painful and inherited.
Hip Dysplasia:
Normal hip joint conformity is lacking because the coxofemoral joint develops incorrectly and laxity occurs. No chemical deficiency is identified as the direct cause in the provided texts. This instability promotes cartilage loss and osteoarthritis over time. Rapid growth and overfeeding can worsen expression of the trait.
Primary Lens Luxation
Normal zonular support of the lens is lacking because inherited weakness and breakdown of the supporting ligaments displace the lens. No nutrient deficiency is identified as the direct cause in the provided texts. Forward lens displacement can raise eye pressure and trigger glaucoma. The condition is painful and potentially blinding.
Cataracts
Normal lens transparency is lacking because the lens becomes opaque and no specific chemical deficiency is identified as the direct cause in the provided texts. In some dogs cataracts may be associated with diabetes mellitus , trauma or chronic eye disease. Loss of transparency reduces light transmission to the retina and impairs vision.
Neuronal Ceroid Lipofuscinosis
Lysosomal function is lacking because inherited mutation causes abnormal storage of autofluorescent lipopigment within nervous tissue. The provided texts do not name a specific missing nutrient or chemical replacement. This cellular storage disorder leads to progressive central nervous system degeneration. Neurologic signs and visual changes can worsen over time.
Progressive Retinal Atrophy:
- There is no cure and management may focus on routine eye exams , environmental adaptation , safety changes and responsible DNA testing of breeding dogs. Dogs with PRA or known risk variants should not be bred.
Primary Lens Luxation:
- Anterior lens luxation is a medical emergency and treatment may involve urgent pressure-lowering medication and surgical lens removal by an ophthalmology specialist. Dogs that develop lens luxation should not be bred.
Hip Dysplasia
- Management may include maintaining a healthy body weight , low-impact exercise , physical therapy , pain relief and in severe cases surgery such as FHO or total hip replacement. Dogs with CHD should not be used for breeding.
Cataracts:
- Early cataracts may not need immediate intervention but progressive cases may require ophthalmology referral and lens removal surgery after retinal testing and ultrasound assessment.
Neuronal Ceroid Lipofuscinosis:
- There is no cure and supportive care may include environmental modification , seizure management and DNA testing to identify affected or carrier lines. Ask professional vets for detailed information.
Progressive Retinal Atrophy :
- 1. Antioxidant supplements can,might be helpful for supporting retinal function and may help delay cataract formation.
Primary Lens Luxation
- 1. No supplement is established as corrective in the provided texts.
Hip Dysplasia
- 1. Glucosamine can,might be helpful for joint support.
2. Omega-3 fatty acids can,might be helpful for reducing joint inflammation.
3. Cartilage protective supplements can,might be helpful for long-term mobility support.
Cataracts
- 1. No supplement is established as curative in the provided texts.
Neuronal Ceroid Lipofuscinosis
- 1. No supplement is established as curative in the provided texts.
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+852 9160 8619
+852 9160 8619